Chronobiology and Obesity by Marta Garaulet, Jose M. Ordovás

By Marta Garaulet, Jose M. Ordovás

Circadian rhythms are such an innate a part of our lives that we
rarely pause to invest why they even exist. probably this is
the this is because within the scientific perform the circadian variability
of hormones, metabolites, physiological behaviors or the
relevance of time within the presence or absence of different
pathologies, were essentially neglected some time past. Some
studies have prompt that the disruption of the circadian
system can be reason for weight problems and manifestations of Metabolic
Syndrome (MetS). Shift-work, sleep-deprivation and
bright-light-exposure at evening are relating to elevated adiposity
(obesity) and occurrence of MetS. Chronobiology and Obesity
examines contemporary advances that experience ended in researchers to discover the
relevant hyperlink which exists among chronobiology and obesity
but, because it is emphasised in the course of the booklet, there's a lot more
that should be explored among the connection

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Additional resources for Chronobiology and Obesity

Sample text

A. Madrid Pérez 23. Vera LM, De Oliveira C, López-Olmeda JF, Ramos J, Mañanós E, Madrid JA, Sánchez-Vázquez FJ (2007) Seasonal and daily plasma melatonin rhythms and reproduction in Senegal sole kept under natural photoperiod and natural or controlled water temperature. J Pineal Res 43:50–55 24. Hardeland R, Madrid JA, Tan DX, Reiter RJ (2012) Melatonin, the circadian multioscillator system and health: the need for detailed analyses of peripheral melatonin signaling. J Pineal Res 52:139–166 25.

In addition, it has been shown that these cytokines also influence energy intake by enhancing insulin and leptin sensitivity. The study of this cytokine offers the possibility to connect the showed interactions among energy intake, sleep behaviour, and circadian system with metabolic alterations. Human studies have indicated that complement proteins, secreted by adipocytes, the so-called Complement-Related Proteins, positively correlated with adiposity, insulin resistance, dyslipidemia, and cardiovascular disease [26].

The beneficial effects of this hormone are predominantly mediated by binding to two cell membrane receptors, adiponectin receptor 1 (ADIPOR1) and adiponectin receptor 2 (ADIPOR2) [15]. Both receptors are also present in adipose tissue, suggesting that adiponectin may have biological effects in adipose tissue in an autocrine/paracrine manner [16]. One of the most controversial adipokines described has been Resistin (resistance to insulin). This adipokine was identified in 2001 as a novel mRNA induced during adipocyte differentiation but down-regulated by thiazolidinediones (TZDs) in vitro [17].

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