Cellular Lipid Metabolism by Sven-Olof Olofsson, Pontus Boström, Jens Lagerstedt (auth.),
By Sven-Olof Olofsson, Pontus Boström, Jens Lagerstedt (auth.), Christian Ehnholm (eds.)
For years lipids have involved mobile biologists and biochemists because of their profound results on cellphone functionality. "Cellular Lipid Metabolism" highlights new ideas and up to date findings, but additionally reports very important discoveries made long ago. remarkable foreign specialists give a contribution thirteen chapters at the genetics, molecular and mobilephone biology of lipids. proposing analyses on the molecular point they display the rules wherein mobile lipid metabolism features. extra, various exciting observations that can't but be defined are pointed out, stimulating the readers to destiny reviews. This ebook presents a useful resource of knowledge for biomedical researchers in strength metabolism, vascular biology, endocrinology and lipidology.
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Taylor and Francis, London, pp 1–15 Olofsson SO, Boren J (2005) Apolipoprotein B: a clinically important apolipoprotein which assembles atherogenic lipoproteins and promotes the development of atherosclerosis. J Intern Med 258:395–410 Olofsson S-O, Bjursell G, Boström K, Carlsson P, Elovson J, Protter AA, Reuben MA, Bondjers G (1987) Apolipoprotein B: structure, biosynthesis and role in the lipoprotein assembly process. Atherosclerosis 68:1–17 1 The Lipid Droplet 25 Olofsson S-O, Asp L, Borén J (1999) The assembly and secretion of apolipoprotein B-containing lipoproteins.
Biochim Biophys Acta 1771:210–227 Dashti N, Gandhi M, Liu X, Lin X, Segrest JP (2002) The N-terminal 1000 residues of apolipoprotein B associate with microsomal triglyceride transfer protein to create a lipid transfer pocket required for lipoprotein assembly. Biochemistry 41:6978–6987 Davidson NO, Shelness GS (2000a) Apolipoprotein B: mRNA editing, lipoprotein assembly, and presecretory degradation. Annu Rev Nutr 20:169–193 Davidson NO, Shelness GS (2000b) APOLIPOPROTEIN B: mRNA editing, lipoprotein assembly, and presecretory degradation.
2000). Furthermore, changes in plasma 24(S)-OHC levels are found in patients with multiple sclerosis (Leoni et al. 2002). Serum 24(S)-OHC concentration apparently reflects the rate of cholesterol turnover in the brain, and it has been suggested as a new diagnostic indicator of diseases affecting the CNS. Hepatic cholesterol-7a-hydroxylase (encoded by CYP7A1) carries out the initial enzymatic step in the major, so-called neutral pathway of bile acid synthesis (reviewed by Björkhem and Eggertsen 2001) .